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Download PDF by Frank J. Dixon (Ed.): Advances in Immunology, Vol. 42

By Frank J. Dixon (Ed.)

ISBN-10: 0120224429

ISBN-13: 9780120224425

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1987). Thus, formal evidence that intact self-cyt c is responsible for elimination of B cells recognizing the /3 bend region of murine cyt c remains elusive. Nonetheless, the likelihood that this phenomenon represents evidence of self-tolerance remains high. A second instance in which a marked disparity exists between the CLONOTYPE REPERTOIRE OF B CELL SUBPOPULATIONS 45 frequencies of responsive cells in the sIg- precursor cell pool and the mature B cell pool, which may be attributable to an environmental antigen, is found in responses to PC in cells from mice bearing the CBA/N immunologic defect.

It was reasoned, therefore, that h-bearing B cells that respond to NP should also respond to carrier conjugates of analogs of NP including NIP. In experiments carried out to assess this point it was found that, indeed, NIP stimulated the same frequency of X-bearing splenic B cells as did NP and, in fact, the proportion of these B cells that bore NPb idiotypic determinants as well as the Ac146 private antigenic determinant was identical to that found when stimulation was carried out with NP. Additionally, the fine specificity as assessed by reactivity of the antibody to hapten analogs was the same in the two populations.

KLINMAN AND PHYLLIS-JEAN LINTON plored concerns the relationship of particular stages of V region gene segment rearrangement with clonal development and expansion. Although at the present time the sequence of molecular events ( D to J h , V h to D h , V, to JL) appears to be emerging (see Section 11), the correlation between each of these events and B cell development has not yet been carefully explored. In particular, it is not yet possible to know the extent of division potential of cells that have undergone each of these molecular events.

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Advances in Immunology, Vol. 42 by Frank J. Dixon (Ed.)

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