By Anish Bhardwaj, Nabil J. Alkayed, Jeffrey R. Kirsch, Richard J. Traystman
Because the 3rd top reason behind demise within the usa, stroke debts for one in each fifteen deaths and is the most important reason behind incapacity within the kingdom. Compiled by means of a popular editorial group, this reference bridges the distance among simple technology and sufferer care protocols, and collects forty three expertly written chapters that diversity from laboratory-based study on animal versions to the development, mechanisms, therapy, and diagnosis of ailments reminiscent of subarachnoid hemorrhage (SAH), intracerebral hemorrhage (ICH), focal ischemic stroke, and international cerebral ischemia.
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Additional resources for Acute Stroke: Bench to Bedside (Neurological Disease and Therapy)
Mayberg MR, Okada T, Bark DH. The role of hemoglobin in arterial narrowing after subarachnoid hemorrhage. J Neurosurg 1990; 72:634–640. 126. Alabadi JA, Salom JB, Torregrosa G, Miranda FJ, Jover T, Alborch E. Changes in the cerebrovascular effects of endothelin-1 and nicardipine after experimental subarachnoid hemorrhage. Neurosurgery 1993; 33:707–714; discussion 714–705. 2 Pathogenesis of Cerebral Aneurysm Growth and Rupture Wesley Hsu, MD, Resident Department of Neurosurgery, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA Richard E.
Whereas vasospasm after endovascular perforation or single hemorrhage was more pronounced in the PCoA, vasospasm after double hemorrhage was more pronounced in the basilar artery. ANIMAL MODELS OF SUBARACHNOID HEMORRHAGE 7 Figure 3 Rat femoral artery model of vasospasm. (A) Artist’s illustration of the surgical technique for induction of vasospasm in the femoral artery of a rat. (B) Microphotograph of a cross-section of the femoral artery of a rat after peri-adventitial blood deposition. Source: From Ref.
Especially of potassium ion concentration in subarachnoid hematoma and its role in cerebral vasospasm. Neurol Med Chir (Tokyo) 1982; 22:805–812. 109. Lacy PS, Earle AM. A small animal model for electrocardiographic abnormalities observed after an experimental subarachnoid hemorrhage. Stroke 1983; 14:371–377. 110. Solomon RA, Antunes JL, Chen RY, Bland L, Chien S. Decrease in cerebral blood flow in rats after experimental subarachnoid hemorrhage: a new animal model. Stroke 1985; 16:58–64. 111. Swift DM, Solomon RA.
Acute Stroke: Bench to Bedside (Neurological Disease and Therapy) by Anish Bhardwaj, Nabil J. Alkayed, Jeffrey R. Kirsch, Richard J. Traystman