By Anish Bhardwaj, Jeffrey R. Kirsch
The medical administration of sufferers with acute mind and spinal twine damage has developed considerably with the arrival of latest diagnostic and healing modalities. Editors Bhardwaj, Ellegala, and Kirsch current Acute mind and Spinal twine Injury , a brand new stand-alone connection with support todayвЂ™s neurologists and neurosurgeons retain abreast of all of the contemporary developments in mind and spinal wire harm. Divided into 5 sections, worrying mind harm, ischemic stroke, intracerebral and subarachnoid hemorrhage, and spinal twine harm, this article bargains the most up-tp-date scientific technology and highlights controversies within the scientific administration of sufferers with acute mind and spinal wire injuries.
Acute mind and Spinal twine Injury :
- each part delineates diagnostic and tracking instruments, pharmacotherapies, and interventional and surgical remedies are covered
- examines and explores lately released laboratory trials and research
- incorporates over 50 diagrams and figures for concise verbal exchange of medical information
Read or Download Acute Brain and Spinal Cord Injury: Evolving Paradigms and Management PDF
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Extra resources for Acute Brain and Spinal Cord Injury: Evolving Paradigms and Management
Importantly, patients may have had surgery for evacuation of a mass lesion. More specifically, the inclusion criteria will be patients who are aged 10 to 65 years with head injury and an abnormal CT scan who require ICP monitoring with raised ICP (>25 mmHg for >1–12 hours) refractory to initial medical treatment measures (48). Criteria meriting exclusion from the study are patients with bilateral fixed and dilated pupils, bleeding diathesis, and devastating injury who are not expected to survive for 24 hours.
Cranioplasty: cosmetic or therapeutic? Surg Neurol 1997; 47(3):238–241. 35. Schiffer J, Gur R, Nisim U, et al. Symptomatic patients after craniectomy. Surg Neurol 1997; 47(3):231–237. 36. Polin RS, Shaffrey ME, Bogaev CA, et al. Decompressive bifrontal craniectomy in the treatment of severe refractory posttraumatic cerebral edema. Neurosurgery 1997; 41(1):84–92 (discussion 92–84). 37. Yang XJ, Hong GL, Su SB, et al. Complications induced by decompressive craniectomies after traumatic brain injury.
Cerebral Blood Flow and CPP A central tenet of cerebrovascular physiology is that a constant supply of metabolic substrates is maintained by a constant CBF over a range of CPPs, a concept known as pressure autoregulation, which is distinct from metabolic autoregulation. CPP is defined as the difference between mean arterial pressure (MAP) and the intracranial pressure (ICP). Normally, CBF is relatively constant in a range of CPPs between 40 and 150 mmHg. A dynamic system of arterial vasoconstriction and dilation exists to preserve CBF and oxygen delivery.
Acute Brain and Spinal Cord Injury: Evolving Paradigms and Management by Anish Bhardwaj, Jeffrey R. Kirsch